Abstract
Multiple sclerosis is an autoimmune disease characterized by inflammatory demylination of the nerves in the central nervous system (brain and spinal cord). Depending on the attacked sites, the disease manifestations vary from limited sensory and/or motoric deficits to wide spread neurological and cognitive deficits. Similar to other autoimmune neurological and non-neurological diseases, the available therapeutic strategies rely on the pharmacological modulation of the immunity, which could be associated with significant side effects.
Depending on the attacked sites, the disease manifestations vary from limited sensory and/or motoric deficits to wide spread neurological and cognitive deficits. Similar to other autoimmune neurological and non-neurological diseases, the available therapeutic strategies rely on the pharmacological modulation of the immunity, which could be associated with significant side effects.
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Correspondence
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Accepted
Published
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